Autor: |
Riabenko DV, Sidorik LL, Fedorkova OM, Bobyk VI, Kovenia TV, Tregubov VS, Danilova VM, Matsuka GKh |
Jazyk: |
ruština |
Zdroj: |
Likars'ka sprava [Lik Sprava] 2001 Mar-Apr (2), pp. 36-9. |
Abstrakt: |
ATP activity of actomyosin in dilated cardiomyopathy (DCMP) was studied together with impact on the above activity of tropomyosin-troponin regulatory complexes recovered from the normal myocardium and cardiac muscle of a DCMP patient. Recordable in DCMP was a striking decline (1.5-fold) in ATP activity of actomyosin. But no significant difference was to be seen in sensitivity to Ca2+ ions of actomyosins obtained from the normal myocardium and cardiac muscle of the DCMP patient. The cardial tropomyosin-troponin regulatory complex from the DCMP patient's myocardium was shown to be endowed with somewhat more manifest activity compared to the analogous complex recovered from the normal myocardium. |
Databáze: |
MEDLINE |
Externí odkaz: |
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