A mutation in the β3 cytoplasmic tail causes variant Glanzmann thrombasthenia by abrogating transition of αIIbβ3 to an active state

Autor: HAUSCHNER, H., MOR‐COHEN, R., SELIGSOHN, U. *, ROSENBERG, N.
Zdroj: In Journal of Thrombosis and Haemostasis February 2012 10(2):289-297
Databáze: ScienceDirect