Liver-chip identifies mitochondrial dysfunction, oxidative stress, and innate immune response as potential pathways of toxicity for the GPR40 agonist TAK-875
Autor: | Jang, K.-J., Otieno, M., Ronxhi, J., Kodella, K., Rubins, J., Simic, D., Singer, M., Lim, H.-K., Lam, W., Chen, J., Petropolis, D., Kulkarni, G., Guzzie-Peck, P., Karalis, K., Hamilton, G. |
---|---|
Zdroj: | In Toxicology Letters 10 October 2018 295 Supplement 1:S129-S130 |
Databáze: | ScienceDirect |
Externí odkaz: |