Liver-chip identifies mitochondrial dysfunction, oxidative stress, and innate immune response as potential pathways of toxicity for the GPR40 agonist TAK-875

Autor: Jang, K.-J., Otieno, M., Ronxhi, J., Kodella, K., Rubins, J., Simic, D., Singer, M., Lim, H.-K., Lam, W., Chen, J., Petropolis, D., Kulkarni, G., Guzzie-Peck, P., Karalis, K., Hamilton, G.
Zdroj: In Toxicology Letters 10 October 2018 295 Supplement 1:S129-S130
Databáze: ScienceDirect