A naturally occurring Tyr143HisαIIb mutation abolishes αIIbβ3 function for soluble ligands but retains its ability for mediating cell adhesion and clot retraction: comparison with other mutations causing ligand-binding defects

Autor: Kiyoi, Teruo, Tomiyama, Yoshiaki *, Honda, Shigenori, Tadokoro, Seiji, Arai, Morio, Kashiwagi, Hirokazu, Kosugi, Satoru, Kato, Hisashi, Kurata, Yoshiyuki, Matsuzawa, Yuji
Zdroj: In Blood 1 May 2003 101(9):3485-3491
Databáze: ScienceDirect