Autor: |
E G, Kolokol'chikova, A A, Pal'tsyn, N V, Panova, A M, Svetukhin, A B, Zemlianoĭ, N V, Chervonskaia, A K, Badikova, L S, Puchkova |
Rok vydání: |
1999 |
Předmět: |
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Zdroj: |
Arkhiv patologii. 61(4) |
ISSN: |
0004-1955 |
Popis: |
Two groups of patients with a grave form of diabetes mellitus type I and II with diabetic foot syndrome have been examined. Group 1 (21 cases) consisted of patients whose foot was amputated. The cause of the necrotic process was ischemia produced by alteration of the arterial vessels aggravated by infection. Patients of group 2 (18 cases) were treated with resultant wound healing and foot preservation. The material was subdivided into two subgroups: 1) tissue from the "infectious" foot with great amount of bacteria and inflammation reaction; 2) tissue from the "ischemic" foot with considerable alterations of microvessels structure. In both groups well developed granulation tissue was observed after the treatment; almost all the cells of this tissue included 3H-uridin and labelling with 3H-thymidine was increased in fibroblasts, endotheliocytes and pericytes. |
Databáze: |
OpenAIRE |
Externí odkaz: |
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