Peri-implant Inflammation Defined by the Implant-Abutment Interface
Autor: | David L. Cochran, Joachim S. Hermann, Nina Broggini, Robert K. Schenk, Daniel Buser, Raúl Uriel Medina, Linda M. McManus |
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Rok vydání: | 2006 |
Předmět: |
Male
0301 basic medicine Pathology medicine.medical_specialty Neutrophils Platform switching Peri Alveolar Bone Loss Dentistry Bone crest Dental Abutments Inflammation Statistics Nonparametric Leukocyte Count 03 medical and health sciences Dogs 0302 clinical medicine medicine Animals Mandibular Diseases Periodontitis 610 Medicine & health General Dentistry Dental alveolus Dental Implants business.industry Chemistry Dental Implantation Endosseous 030206 dentistry 030104 developmental biology Dental Prosthesis Design Crest Implant medicine.symptom business Implant abutment |
Zdroj: | Journal of Dental Research. 85:473-478 |
ISSN: | 1544-0591 0022-0345 |
DOI: | 10.1177/154405910608500515 |
Popis: | An implant-abutment interface at the alveolar bone crest is associated with sustained peri-implant inflammation; however, whether magnitude of inflammation is proportionally dependent upon interface position remains unknown. This study compared the distribution and density of inflammatory cells surrounding implants with a supracrestal, crestal, or subcrestal implant-abutment interface. All implants developed a similar pattern of peri-implant inflammation: neutrophilic polymorphonuclear leukocytes (neutrophils) maximally accumulated at or immediately coronal to the interface. However, peri-implant neutrophil accrual increased progressively as the implant-abutment interface depth increased, i.e., subcrestal interfaces promoted a significantly greater maximum density of neutrophils than did supracrestal interfaces (10,512 ± 691 vs. 2398 ± 1077 neutrophils/mm2). Moreover, inflammatory cell accumulation below the original bone crest was significantly correlated with bone loss. Thus, the implant-abutment interface dictates the intensity and location of peri-implant inflammatory cell accumulation, a potential contributing component in the extent of implant-associated alveolar bone loss. |
Databáze: | OpenAIRE |
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