Iodine-induced thyrotoxic hypokalemic paralysis after ingestion ofSalicornia herbace
| Autor: | Dong Jun Park, Seong Eun Yun, Kyungo Hwang, Eun Jin Bae, Yeojin Kang, Ha Nee Jang, Hyun Seop Cho, Se-Ho Chang |
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| Rok vydání: | 2013 |
| Předmět: |
Male
medicine.medical_specialty Flaccid paralysis Potassium Hypokalemic Periodic Paralysis chemistry.chemical_element Chenopodiaceae Critical Care and Intensive Care Medicine Iodine Thyroid function tests Gastroenterology Eating Internal medicine medicine Paralysis Humans Ingestion medicine.diagnostic_test business.industry Periodic paralysis General Medicine Middle Aged medicine.disease Hypokalemia Thyrotoxicosis Endocrinology chemistry Nephrology Dietary Supplements medicine.symptom business |
| Zdroj: | Renal Failure. 36:461-463 |
| ISSN: | 1525-6049 0886-022X |
| DOI: | 10.3109/0886022x.2013.868296 |
| Popis: | A 56-year-old Korean man visited to emergency room due to paroxysmal flaccid paralysis in his lower extremities. There was no family or personal history of periodic paralysis. His initial potassium levels were 1.8 mmol/L. The patient had been taking Salicornia herbacea for the treatment of diabetes and hypertension. Results of a thyroid function test were as follows: T3 = 130.40 ng/dL, TSH = 0.06 mIU/L, and free T4 = 1.73 ng/dL. A thyroid scan exhibited a decreased uptake (0.6%). His symptoms clearly improved and serum potassium levels increased to 4.4 mmol/L by intravenous infusion of only 40 mmol of potassium chloride. Eight months after the discontinuation of only Salicornia herbacea, the patient’s thyroid function tests were normalized. Large amounts of iodine can induce hypokalemic thyrotoxic paralysis and it may be necessary to inquire about the ingestion of iatrogenic iodine compounds, such as Salicornia herbacea. |
| Databáze: | OpenAIRE |
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