Citrus as a molecular contact point for co-evolution of Alternaria pathogens
| Autor: | Syu Matsuoka, Mikihiro Yamamoto, Kazuya Ichimura, Motoichiro Kodama, Kouhei Ohtani, Susumu Mochizuki, Kenji Gomi, Mai Katsumoto, Takashi Tsuge, Kazuya Akimitsu, Chika Igarashi, Sawa Tanaka, Takuya Shimagami |
|---|---|
| Rok vydání: | 2016 |
| Předmět: |
0106 biological sciences
0301 basic medicine biology Toxin Rough lemon food and beverages Plant Science Alternaria biology.organism_classification medicine.disease_cause 01 natural sciences Alternaria alternata 03 medical and health sciences chemistry.chemical_compound 030104 developmental biology chemistry Botany Gene cluster otorhinolaryngologic diseases medicine Genetics Leaf spot Mycotoxin Gene 010606 plant biology & botany |
| Zdroj: | Physiological and Molecular Plant Pathology. 95:93-96 |
| ISSN: | 0885-5765 |
| DOI: | 10.1016/j.pmpp.2016.01.005 |
| Popis: | Alternaria black rot, Alternaria leaf spot of rough lemon, and Alternaria brown spot of tangerines are three major citrus Alternaria pathogens. Citrus could be considered as a molecular contact point for host-selective toxin (HST)-mediated co-evolution of these Alternaria pathogens and susceptibility in the field. ACR-toxin is an HST produced by the rough lemon pathotype, and the target site of the toxin was identified as rough lemon mitochondria. The biosynthetic gene cluster for ACR-toxin production is on the 1.5 Mb-chromosome of the rough lemon pathotype. Another gene cluster for ACT-toxin production is located on the 1.9 Mb-chromosome of the tangerine pathotype. These TOX genes shown to have a role in ACR- or ACT-toxin biosynthesis by using gene disruption and silencing. |
| Databáze: | OpenAIRE |
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