Phylogenetic background and carriage of pathogenicity island-like domains in relation to antibiotic resistance profiles among Escherichia coli urosepsis isolates
| Autor: | Stéphane Bonacorsi, Dominique Barraud, Philippe Bidet, Martine Bingen-Bidois, Edouard Bingen, Véronique Houdouin |
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| Rok vydání: | 2006 |
| Předmět: |
Microbiology (medical)
Genomic Islands Nalidixic acid Virulence Factors Virulence Microbial Sensitivity Tests Biology medicine.disease_cause Polymerase Chain Reaction Microbiology Nalidixic Acid Ribotyping Anti-Infective Agents Sepsis Ampicillin Drug Resistance Bacterial Trimethoprim Sulfamethoxazole Drug Combination Genotype Escherichia coli medicine Humans Pharmacology (medical) Escherichia coli Infections Phylogeny Antibacterial agent Pharmacology Genetics Pathogenicity island Infectious Diseases Urinary Tract Infections medicine.drug |
| Zdroj: | Journal of Antimicrobial Chemotherapy. 58:748-751 |
| ISSN: | 1460-2091 0305-7453 |
| Popis: | We studied 100 well-characterized E. coli blood isolates from patients with urosepsis for their susceptibility to nalidixic acid, ampicillin and trimethoprim-sulfamethoxazole, according to prevalence of virulence factors, phylogenetic groups and subgroups, PAI II(J96)-like domains (determined by physical linkage of cnf1, hly and hra) and PAI I(CFT073)-like domains (determined by physical linkage of papGII to the hly locus). Nalidixic acid resistance was associated with a lower prevalence of sfa/foc, K1 antigen, pathogenicity island II(J96)-like domains, subgroup B2/I and a shift towards group A. |
| Databáze: | OpenAIRE |
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