Recovery processes after repeated supramaximal exercise at the altitude of 4,350 m
| Autor: | Jean-Paul Richalet, Daniel Biou, Murielle Letournel, Denis Deberne, Jenny Vaysse, Paul Robach, Jean-Pierre Herry |
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| Rok vydání: | 1997 |
| Předmět: |
Adult
Male medicine.medical_specialty Physiology Physical exercise Veins Cardiovascular Physiological Phenomena Oxygen Consumption Physiology (medical) medicine Humans Lactic Acid Exercise business.industry Pulmonary Gas Exchange Altitude Effects of high altitude on humans Hypoxia (medical) Hydrogen-Ion Concentration Oxygen uptake Surgery Anesthesia Physical Endurance Female Bicycle ergometer Gases medicine.symptom business Blood ph |
| Zdroj: | Journal of applied physiology (Bethesda, Md. : 1985). 82(6) |
| ISSN: | 8750-7587 |
| Popis: | Robach, Paul, Daniel Biou, Jean-Pierre Herry, Denis Deberne, Murielle Letournel, Jenny Vaysse, and Jean-Paul Richalet. Recovery processes after repeated supramaximal exercise at the altitude of 4,350 m. J. Appl. Physiol. 82(6): 1897–1904, 1997.—We tested the hypothesis that prolonged exposure to high altitude would impair the restoration of muscle power during repeated sprints. Seven subjects performed two 20-s Wingate tests (WT1 and WT2) separated by 5 min of recovery, at sea level (N) and after 5–6 days at 4,350 m (H). Mean power output (MPO) and O2 deficit were measured during WT. O2 uptake (V˙o 2) and ventilation (V˙e) were measured continuously. Blood velocity in the femoral artery (FBV) was recorded by Doppler ultrasound during recovery. Arterialized blood pH and concentrations of bicarbonate ([[Formula: see text]]), venous plasma lactate ([La−]), norepinephrine ([NE]), and epinephrine ([Epi]) were measured before and after WT1 and WT2. MPO decreased between WT1 and WT2 by 6.9% in N ( P < 0.05) and by 10.7% in H ( P < 0.01). H did not further decrease MPO. O2 deficit decreased between WT1 and WT2 in H only ( P < 0.01). PeakV˙o 2 after WT was reduced by 30–40% in H ( P < 0.01), but excess postexercise O2 consumption was not significantly lowered in H. During recovery in H compared with N, V˙e, exercise-induced acidosis, and [NE] were higher, [Epi] tended to be higher, [La−] was not altered, and [[Formula: see text]] and FBV were lower. The similar [La−] accumulation was associated with a higher exercise-induced acidosis and a larger increase in [NE] in H. We concluded from this study that prolonged exposure to high altitude did not significantly impair the restoration of muscle power during repeated sprints, despite a limitation of aerobic processes during early recovery. |
| Databáze: | OpenAIRE |
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