Pathophysiology of supraventricular tachycardia
| Autor: | M. Anderson, A. J. Camm, C. F. Shakespeare |
|---|---|
| Rok vydání: | 1993 |
| Předmět: |
Tachycardia
medicine.medical_specialty Pre-Excitation Mahaim-Type Heart Conduction System Internal medicine Atrial Fibrillation Heart rate Tachycardia Supraventricular Animals Humans Tachycardia Atrioventricular Nodal Reentry Medicine cardiovascular diseases Atrium (heart) business.industry Atrial fibrillation Reentry medicine.disease Atrioventricular node medicine.anatomical_structure Atrial Flutter cardiovascular system Cardiology Wolff-Parkinson-White Syndrome Supraventricular tachycardia medicine.symptom Cardiology and Cardiovascular Medicine business Atrial flutter |
| Zdroj: | European Heart Journal. 14:2-8 |
| ISSN: | 1522-9645 0195-668X |
| DOI: | 10.1093/eurheartj/14.suppl_e.2 |
| Popis: | Supraventricular tachycardia is due to altered automaticity, triggered activity or most commonly, reentry. Atrial flutter is most frequently due to reentry with counterclockwise activation in the right atrium with the left atrium acting as a bystander. The small wavefronts demonstrated during atrial fibrillation are less random than was previously thought. In the study of atrioventricular (AV) nodal reentrant tachycardia, recent evidence suggests that multiple pathways involving intranodal and perinodal tissue are probably involved. In the Wolff-Parkinson-White (WPW) syndrome, accessory pathways near the AV node have been demonstrated with decremental properties, suggesting conduction through accessory AV nodal tissue. The mechanism for atrial fibrillation in the WPW syndrome is poorly understood, but there is evidence that accessory pathways are branched and may themselves support micro-reentry. In Mahaim nodoventricular pathways, the atrium is not essential for reentry, unlike atriofascicular reentrant circuits. Atriofascicular pathways consist of AV node-like tissue and are usually located in the right atrial free wall. |
| Databáze: | OpenAIRE |
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