| Autor: |
Lotfi Chouchane, Francesco M. Marincola, Ena Wang, Edith Mathiowitz, Stacia Furtado, Salah Gehani, Issam Al-Bozom, Joel Malek, Idil I. Aigha, Dhanya Kizhakayil, Shahinaz Bedri, Konduru S. Sastry, Maria L. Ascierto, Eman K. Al-Azwani, Sasha Bakhru, Shoba P. DSouza, Jingxuan Shan |
| Rok vydání: |
2023 |
| Popis: |
PDF file - 655K, Supplementary Figure 1. TNRC9 promoted cell proliferation in MDA-MB-231 breast cancer cells. Supplementary Figure 2. shRNA-mediated knockdown of TNRC9. Supplementary Figure 3. H&E staining of tumors (10�Magnification) from xenograft nude mice subcutaneously injected with control ZR-75-1cells (upper row I � V) were compared with those injected with TNRC9-depeltion ZR-75-1 cells (lower row VI � VIII, no palpable tumor in two mice). �-� represents 400μm. Supplementary Figure 4. Knocking-down TNRC9 sensitizes cancer cells to apoptosis signal. Supplementary Figure 5. BRCA1 and TNRC9 expression in TNRC9 abrogated and control cells. Supplementary Figure 6. The expression profile of TNRC9 knockdown MCF-7 cells. Supplementary Figure 7. In silico analysis of TNRC9 and BRCA1 gene expression among ovarian cancer patients. Supplementary Figure 8. The effects of TNRC9 on luciferase activity driven by BRCA1 promoter. Supplementary Table 1 The list of transcripts significantly altered in TNRC9 knockdown MCF-7 cells comparing to control MCF-7 cells |
| Databáze: |
OpenAIRE |
| Externí odkaz: |
|
