Degenerated lens epithelial cells in rabbit and human eyes after intraocular lens implantation
Autor: | Yoshiji Kawashima, Michio Kimura, Yoshitaka Ohnishi, Shizuya Saika, Akio Yamanaka, Kiyomi Ohkawa, Takeshi Miyamoto, Akira Ooshima, Shunsaku Ohmi |
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Rok vydání: | 1998 |
Předmět: |
medicine.medical_specialty
genetic structures medicine.medical_treatment Lens Capsule Crystalline Intraocular lens Cataract Lens Implantation Intraocular Ophthalmology medicine Animals Humans Vitreous surgery Posterior capsule opacification Aged Lens capsule Phacoemulsification business.industry Capsule Epithelial Cells eye diseases Sensory Systems Posterior capsule medicine.anatomical_structure Lens (anatomy) Female Surgery Rabbits sense organs business Cell Division Intracellular organelles |
Zdroj: | Journal of Cataract and Refractive Surgery. 24:1396-1398 |
ISSN: | 0886-3350 |
DOI: | 10.1016/s0886-3350(98)80236-8 |
Popis: | Purpose: To evaluate the role of lens epithelial cells (LECs) in posterior capsule opacification. Setting: Departments of Ophthalmology and Pathology, Wakayama Medical College, Department of Anatomy, Kansai Shinkyu College, and Department of Ophthalmology, Kobe Kaisei Hospital, Japan. Methods: We examined the presence of degenerated LECs on the capsules of the eyes of rabbits and a patient after intraocular lens (IOL) implantation. Phacoextraction of a crystalline lens and IOL implantation were done in 5 albino rabbits under general anesthesia. The animals were killed after 2 months. Lens capsules were removed and fixed. During vitreous surgery, a lens capsule with an IOL was removed from a patient. Ultrathin sections of specimens were studied by transmission electron microscopy. Results: Presumed LECs proliferated between the posterior capsule and the IOL in association with collagenous matrix, Debris from the degenerated cells and destroyed intracellular organelles was also seen. Conclusion: Lens epithelial cells proliferating on the posterior capsule cannot survive indefinitely. J Cataract Refract Surg 1998; 24:1396-1398 |
Databáze: | OpenAIRE |
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