Myopalladin is upregulated in dilated cardiomyopathies patients and myopalladin knockout mice develop cardiac dilation and dysfunction following pressure overload

Autor:	Roman S. Polishchuk, P. Carullo, R. Crispino, Chiara Tesi, D.L. Yamamoto, M.L. Bang, M.C. Filomena, Jianlin Zhang, Beatrice Scellini, R. Knöll, Corrado Poggesi, Nicoletta Piroddi
Rok vydání:	2018
Předmět:	Pressure overload Myopalladin sarcomeric protein cardiomyopathy medicine.medical_specialty Downregulation and upregulation business.industry Internal medicine Knockout mouse medicine Cardiology Dilation (morphology) Cardiology and Cardiovascular Medicine business Molecular Biology
Zdroj:	Journal of Molecular and Cellular Cardiology. 120:43
ISSN:	0022-2828
Databáze:	OpenAIRE
Externí odkaz:	https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70f61e160dc77b47680e1f0d83b01b80 https://doi.org/10.1016/j.yjmcc.2018.05.129 Zobrazit plný text záznamu Full Text from ScienceDirect