Pulmonary Lesions in Dogs with Experimental and Naturally Occurring Toxoplasmosis
| Autor: | Charles C. Capen, Clarence R. Cole |
|---|---|
| Rok vydání: | 1966 |
| Předmět: |
Lung Diseases
0301 basic medicine Pathology medicine.medical_specialty Necrosis Fibrin 03 medical and health sciences Dogs 0302 clinical medicine medicine Animals Dog Diseases Distemper Respiratory system Animal species Lung 030102 biochemistry & molecular biology biology business.industry General Medicine medicine.disease Toxoplasmosis respiratory tract diseases Pneumonia Toxoplasmosis Animal medicine.anatomical_structure 030220 oncology & carcinogenesis Immunology biology.protein Lymph medicine.symptom business |
| Zdroj: | Pathologia veterinaria. 3:40-63 |
| ISSN: | 0031-2975 |
| DOI: | 10.1177/030098586600300103 |
| Popis: | Toxoplasmagondii has been recognized as a cause of disease for several decades in human beings and many animal species. The clinical signs of both experimental and naturally occurring toxoplasmosis in the dog often indicate a severe involvement of the respiratory system. MELLO~~ described pulmonary hyperemia and focal pneumonia in the initial report of naturally occurring canine toxoplasmosis. Subsequently, numerous reports of canine toxoplasmosis have appeared in the literature. Pulmonary lesions reported in dogs with the naturally occurring disease include miliary necrosis and fibrin exudation, fibrinous pneumonia, bronchopneumonia, focal pneumonia, focal granulomas, enlarged bronchial lymph nodes, and T. gondii in lung smears. COHRS and CARS TENS EN^ reported a severe dyspnea in experimentally infected dogs. The most striking and frequently encountered lesions were found in the lungs and consisted of focal necrosis and fibrinous pneumonia. VALL~E et al.36 described a bronchopneumonia with focal necrosis in young dogs experimentally infected with T. gondii by intratracheal and intraperitoneal routes. The objectives of ths investigation were to: (1) describe the lesions produced by T. gondii in the lungs of dogs, (2) compare the disease induced by respiratory exposure to that following other routes of infection and (3) compare naturally occurring toxoplasmosis with the experimental infection. |
| Databáze: | OpenAIRE |
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