Lung-Function Trajectories Leading to Chronic Obstructive Pulmonary Disease
| Autor: | Stefano Guerra, Hans Petersen, Yohannes Tesfaigzi, Peter Schnohr, Peter Lange, Fernando D. Martinez, Caroline A. Owen, Akshay Sood, Jørgen Vestbo, Pablo Martínez-Camblor, Victor Pinto-Plata, Bartolome R. Celli, Joan B. Soriano, Alvar Agusti, Jacob Louis Marott, Miguel Divo, Gorm B. Jensen, Paula Meek, Rosa Faner |
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| Přispěvatelé: | Universitat de Barcelona, UAM. Departamento de Medicina, Instituto de Investigación del Hospital de La Princesa (IP) |
| Předmět: |
Adult
Male Pediatrics medicine.medical_specialty Pulmonary Disease Chronic Obstructive/etiology Medicina Offspring Population Cohort Studies Malalties de l'aparell respiratori Pulmonary Disease Chronic Obstructive Young Adult Forced Expiratory Volume Smoking/adverse effects Humans Medicine Young adult Chronic obstructive pulmonary diseases education Lung Malalties pulmonars obstructives cròniques Aged education.field_of_study COPD Framingham Risk Score business.industry Smoking Pulmó Respiratory organs diseases FEV General Medicine Middle Aged respiratory system Chort medicine.disease respiratory tract diseases medicine.anatomical_structure Cohort Disease Progression Female Genesis of COPD Pulmonary disease business Cohort study circulatory and respiratory physiology |
| Zdroj: | Recercat. Dipósit de la Recerca de Catalunya instname Biblos-e Archivo. Repositorio Institucional de la UAM Lange, P, Celli, B, Agustí, A, Jensen, G B, Divo, M, Faner, R, Guerra, S, Marott, J L, Martinez, F D, Martinez-Camblor, P, Meek, P, Owen, C A, Petersen, H, Pinto-Plata, V, Schnohr, P, Sood, A, Soriano, J B, Tesfaigzi, Y & Vestbo, J 2015, ' Lung-function trajectories leading to chronic obstructive pulmonary disease ', The New England Journal of Medicine, vol. 373, no. 2, pp. 111-122 . https://doi.org/10.1056/NEJMoa1411532 Dipòsit Digital de la UB Universidad de Barcelona |
| DOI: | 10.1056/NEJMoa1411532 |
| Popis: | BACKGROUND Chronic obstructive pulmonary disease (COPD) is thought to result from an accelerated decline in forced expiratory volume in 1 second (FEV1) over time. Yet it is possible that a normal decline in FEV1 could also lead to COPD in persons whose maximally attained FEV1 is less than population norms. METHODS We stratified participants in three independent cohorts (the Framingham Offspring Cohort, the Copenhagen City Heart Study, and the Lovelace Smokers Cohort) according to lung function (FEV1 ≥80% or |
| Databáze: | OpenAIRE |
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