Hyperkalemic neuromyopathy in Addison's disease
| Autor: | Robert H. Williams, Richard H. Pollen |
|---|---|
| Rok vydání: | 1960 |
| Předmět: |
medicine.medical_specialty
Hyperkalemia Potassium chemistry.chemical_element POTASSIUM RETENTION Addison Disease Internal medicine Paralysis medicine Adrenal insufficiency Humans business.industry Muscle weakness General Medicine Neuromuscular Diseases medicine.disease Endocrinology chemistry Hypoadrenocorticism Familial Addison's disease medicine.symptom business Hormone Adrenal Insufficiency |
| Zdroj: | The New England journal of medicine. 263 |
| ISSN: | 0028-4793 |
| Popis: | ALTHOUGH a deficiency of adrenocortical hormones is commonly associated with potassium retention, clinical manifestations of hyperkalemia in untreated Addison's disease are quite rare.1 However, a Landry type of rapidly ascending muscular weakness that leads to flaccid quadriplegia but tends to preserve cerebration and cranial-nerve function is occasionally observed with the high serum potassium levels accompanying renal insufficiency.2 3 4 These patients die from the neuromyocardial effect of excess potassium ion. Although it is recognized that the administration of potassium salts to patients with Addison's disease5 , 6 and even to normal subjects7 may induce profound muscle weakness, cramps and vivid olfactory and cutaneous paresthesias, . . . |
| Databáze: | OpenAIRE |
| Externí odkaz: |
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