Optic nerve compression by carotid arteries in low-tension glaucoma
| Autor: | Isaac Gutman, Michael Blumenthal, Shlomo Melamed, Isaac Ashkenazi |
|---|---|
| Rok vydání: | 1993 |
| Předmět: |
Carotid Artery Diseases
Male medicine.medical_specialty genetic structures Fundus Oculi Carotid arteries Eye disease Glaucoma Ocular Hypotension Cellular and Molecular Neuroscience Ectasia Ophthalmology medicine Humans Intraocular Pressure Aged Aged 80 and over Optic canal business.industry Nerve Compression Syndromes Calcinosis Optic Nerve Middle Aged medicine.disease eye diseases Sensory Systems Surgery Vasodilation Low Tension Glaucoma medicine.anatomical_structure Optic nerve Female sense organs Visual Fields Tomography X-Ray Computed business Orbit Calcification |
| Zdroj: | Graefe's Archive for Clinical and Experimental Ophthalmology. 231:711-717 |
| ISSN: | 1435-702X 0721-832X |
| DOI: | 10.1007/bf00919286 |
| Popis: | Low-tension glaucoma (LTG) is manifested by glaucomatous optic nerve damage and visual field loss despite normal intraocular pressure (IOP). We describe 62 patients with classical signs of LTG. Computed tomography (CT) was performed in all patients. In 56 of the patients (90.3%), pathology of the intracavernous carotid arteries adjacent to the intracranial opening of the optic canal could be demonstrated. In 28 patients (45.2%) a clear asymmetry of the optic nerve cupping was found and could be correlated with the severity of the carotid artery pathology. A control group of 24 age-matched patients included five (20.8%) with intracavernous carotid artery calcification and only one (4.2%) with intracavernous ectasia. We suggest that calcification, dilatation and ectasia of the carotid artery into the optic canal may play an important role in the pathogenesis of many cases of LTG. The close proximity of the carotid artery to the optic nerve at this location may result in compressive neuropathy with subsequent glaucomatous damage of the optic nerve head. |
| Databáze: | OpenAIRE |
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