Licorice-induced apparent mineralocorticoid excess causing persistent hypertension and hypokalemia
| Autor: | G Makar, N Awad, S R Burroughs, V Burroughs, P Ravi |
|---|---|
| Jazyk: | angličtina |
| Rok vydání: | 2020 |
| Předmět: |
medicine.medical_specialty
Aldosterone Endocrine and Autonomic Systems business.industry Endocrinology Diabetes and Metabolism Metabolic alkalosis Chronic ingestion Case Report medicine.disease Hypokalemia chemistry.chemical_compound Endocrinology chemistry Internal medicine APPARENT MINERALOCORTICOID EXCESS medicine Tea intake medicine.symptom Hyponatremia LICORICE INGESTION business |
| Zdroj: | Acta Endocrinol (Buchar) |
| Popis: | Chronic ingestion of licorice is known to cause numerous metabolic and electrolyte disturbances. Severe hyponatremia, hypertension, and hypokalemia as well as metabolic alkalosis are amongst the most common consequences of chronic ingestion resulting in an apparent mineralocorticoid excess (AME). Treatment predominantly consists of cessation of licorice ingestion, potassium replenishment and aldosterone antagonists. Given the potentially lethal effects of chronic licorice ingestion, clinicians should be made aware of the presentation of AME and the proper management. We present the rare case of a 62-year-old male with licorice-induced apparent mineralocorticoid excess secondary to excessive licorice tea intake. Initial presentation included severe hypokalemia of 2.2mmol/L and hypertension of 180/110mmHg, while eunatremic (Na, 144meq/L). |
| Databáze: | OpenAIRE |
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