LATE RISE IN SGPT AFTER ACUTE MYOCARDIAL INFARCTION
| Autor: | Urbain Säwe, L. R. Erhardt, A. Sjögren |
|---|---|
| Rok vydání: | 2009 |
| Předmět: |
Male
medicine.medical_specialty Time Factors Myocardial Infarction Serum enzymes Sulfobromophthalein Excretion Hepatic function Electrocardiography Liver Function Tests Internal medicine Internal Medicine medicine Humans Aspartate Aminotransferases cardiovascular diseases Myocardial infarction Aged L-Lactate Dehydrogenase biology medicine.diagnostic_test business.industry Alanine Transaminase Middle Aged Alkaline Phosphatase medicine.disease Surgery Alanine transaminase Acute Disease biology.protein Cardiology Alkaline phosphatase Female Liver function tests business Follow-Up Studies |
| Zdroj: | Acta Medica Scandinavica. 196:47-51 |
| ISSN: | 0001-6101 |
| DOI: | 10.1111/j.0954-6820.1974.tb00966.x |
| Popis: | The present study draws attention to a late rise in SGPT following acute myocardial infarction (AMI) beginning about 4 days after the SGOT max. This observation is illustrated by an investigation of 7 patients, selected because of this finding, and 7 controls, also with an AMI, but without this enzyme pattern. Peak SGOT and LDH enzyme levels during the acute phase were similar in the two groups. The late SGPT rise was associated with high serum alkaline phosphatases 2 weeks after the AMI, but not 5–9 months later. Two weeks after onset of symptoms BSP excretion was commonly impaired but failed to separate the two groups. In contrast to the controls, BSP excretion was significantly improved at reinvestigation 5–9 months after the AMI in the group with the SGPT rise. 17) and can at times jeopardise the serum enzyme diagnosis of AMI (4, 5). The present investigation of patients with AMI was initiated by repeated observations of a late rise in SGPT beginning about a week after onset of symptoms and exceeding simultaneous SGOT values. This rise was usually not accompanied by a rise in SGOT and was therefore assumed not to reflect reinfarction, nor was there any clinical evidence of this being the case. In an attempt to clarify this enzyme rise a further evaluation was made of hepatic function in 14 patients with AMI, 7 with and 7 without this enzyme rise. |
| Databáze: | OpenAIRE |
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