Critical features of periodontal flaps with regard to blood clot stability: A review
| Autor: | Yasser Riaz Malik, Muhammad Sohail Zafar, Mohid Abrar Lone, Flavio Pisani, Muhammad Saad Shaikh |
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| Rok vydání: | 2021 |
| Předmět: |
0301 basic medicine
Periodontium Periodontal tissue Gingiva Medicine (miscellaneous) Connective tissue Adhesion (medicine) Dentistry General Biochemistry Genetics and Molecular Biology 03 medical and health sciences 0302 clinical medicine medicine Humans General Dentistry Gingival recession Wound Healing business.industry Regeneration (biology) Mucogingival surgery Thrombosis 030206 dentistry medicine.disease 030104 developmental biology medicine.anatomical_structure Connective Tissue medicine.symptom business Wound healing |
| Zdroj: | Journal of oral biosciences. 63(2) |
| ISSN: | 1880-3865 |
| Popis: | Background Wound healing is a multifactorial procedure involving different cell types and biological mediators. The principles of wound healing are also applicable to periodontal tissues. The formation and stability of blood clots play a vital role in successful healing of wounds in periodontal tissues. The aim of the present review was to highlight the vital factors of periodontal flaps associated with blood clot stability. Highlight The data on periodontal regeneration and wound healing have evolved greatly in light of several factors, including space for blood clots and blood clot stabilization. In periodontal osseous defects, the stability of blood clots seems critical to wound healing. If mechanical forces can be managed by wound stabilization, the gingival flap-tooth root interface may show connective tissue repair. However, compromised adhesion is susceptible to mechanical forces and can cause wound breakage and epithelialization. Conclusion The presence of a thick blood clot may hinder the plasmatic circulation between the recipient bed and graft during the initial stage of healing, which is critical in cases of mucogingival surgery. Root conditioning can also determine the healing consequence by enhancing blood clot adhesion. |
| Databáze: | OpenAIRE |
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