Arterial Blood Gases During Diving in Elite Apnea Divers
| Autor: | S. Hamich, Jürgen M. Steinacker, Peter Radermacher, Klaus Paulat, Claus-Martin Muth, Enrico Calzia, R. Schabana, A. Pittner |
|---|---|
| Rok vydání: | 2003 |
| Předmět: |
Adult
Diving Partial Pressure Vital Capacity Poison control Physical Therapy Sports Therapy and Rehabilitation Co2 storage Buccal pumping medicine Humans Orthopedics and Sports Medicine Acid-Base Equilibrium Pulmonary Gas Exchange business.industry Apnea Gas exchange Partial pressure Carbon Dioxide respiratory tract diseases Oxygen Control of respiration Anesthesia Arterial blood Blood Gas Analysis medicine.symptom business human activities |
| Zdroj: | International Journal of Sports Medicine. 24:104-107 |
| ISSN: | 1439-3964 0172-4622 |
| DOI: | 10.1055/s-2003-38401 |
| Popis: | Elite apnea divers have considerably extended the limits of dive depth and duration but the mechanisms allowing humans to tolerate the compression- and decompression-induced changes in alveolar gas partial pressures are still not fully understood. Therefore we measured arterial blood gas tensions and acid-base-status in two elite apnea divers during simulated wet dives lasting 3 : 55 and 5 : 05 minutes, respectively. Arterial pO2 followed the compression-(from 13.8/16.9 kPa before the dive to 30 kPa at the start of the bottom time) and decompression-induced (from 13.7/21.0 kPa to 3.3/4.9 kPa immediately after surfacing) variations of ambient pressure, while the arterial pCO2 remained within the physiologic range (3.0/3.9 kPa before diving vs. 5.7/5.9 kPa at the end of the bottom time), probably due to the CO2 storage capacity of the blood. These findings may help to explain why humans can sustain deep and long apnea dives without major increases in respiratory drive. Language: en |
| Databáze: | OpenAIRE |
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