Severe hypokalemic paralysis and rhabdomyolysis occurring after binge eating in a young bodybuilder
| Autor: | Dae-Hong Jeon, Hyun Seop Cho, Hee Jung Park, Dong Jun Park, Se-Ho Chang, Kyungo Hwang, Tae Won Lee, Ha Nee Jang, Eunjin Bae |
|---|---|
| Jazyk: | angličtina |
| Rok vydání: | 2017 |
| Předmět: |
Adult
Male Pediatrics medicine.medical_specialty Weight Lifting 030209 endocrinology & metabolism macromolecular substances paralysis Hypokalemic paralysis 03 medical and health sciences 0302 clinical medicine medicine hypokalemia Humans 030212 general & internal medicine Clinical Case Report Bulimia Binge eating business.industry General Medicine medicine.disease Hypokalemia Anesthesia rhabdomyolysis medicine.symptom business Paralysis Hyperkalemic Periodic Rhabdomyolysis Research Article |
| Zdroj: | Medicine |
| ISSN: | 1536-5964 0025-7974 |
| Popis: | Rationale: Severe hypokalemia can be a potentially life-threatening disorder and is associated with variable degrees of skeletal muscle weakness. Patient concerns: We report a case of severe hypokalemic paralysis and rhabdomyolysis in a 28-year-old bodybuilder. He was admitted to the emergency room due to progressive paralysis in both lower extremities, which had begun 12 hours earlier. He was a bodybuilder trainer and had participated in a regional competition 5 days earlier. He went on a binge, consuming large amounts of carbohydrates over 4 days, resulting in a gain of 10 kg in weight. Diagnoses: He had no family history of paralysis and this was his first attack. He strongly denied drug abuse, such as anabolic steroids, thyroid and growth hormone, and diuretics. Neurological examinations revealed symmetrical flaccid paralysis in his lower extremities, but the patient was alert and his sensory system was intact. His initial serum potassium and phosphate level was 1.8 mmol/L and 1.4 mg/dL, respectively. The calculated transtubular potassium gradient (TTKG) was 2.02. His thyroid function was normal. Interventions and Outcomes: Serum potassium levels increased to 3.8 mmol/L with intravenous infusion of about 50 mmol of potassium chloride over 20 hours. Outcomes: His muscular symptoms improved progressively and he was discharged from the hospital 7 days after admission on foot. He was followed in our outpatient clinic, without recurrence. Lessons: Physicians should keep in mind that large intakes of food during short periods can provoke hypokalemic paralysis and rhabdomyolysis, especially in bodybuilders. |
| Databáze: | OpenAIRE |
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