Hemodynamic and catecholamine responses associated with extracorporeal shock wave lithotripsy
| Autor: | Jonathan Moss, Harry W. Linde, Michael F. Roizen, Rahim Behnia, John B. Graham |
|---|---|
| Rok vydání: | 1990 |
| Předmět: |
Tachycardia
Adult Male Mean arterial pressure Cardiac output Epinephrine medicine.medical_treatment Hemodynamics Anesthesia General Norepinephrine Lithotripsy Immersion medicine Humans business.industry Middle Aged Extracorporeal shock wave lithotripsy Anesthesiology and Pain Medicine Isoflurane Anesthesia Hypertension Catecholamine Female medicine.symptom business medicine.drug |
| Zdroj: | Journal of clinical anesthesia. 2(3) |
| ISSN: | 0952-8180 |
| Popis: | Patients undergoing extracorporeal shock wave lithotripsy (ESWL) for nephrolithiasis are anesthetized and immersed in water in a semisitting position. Hypertension and tachycardia have been reported to accompany ESWL, and it was hypothesized that those problems were a result of adrenal medullary release of epinephrine or norepinephrine. Therefore, the effects of ESWL on cardiovascular variables and circulating epinephrine and norepinephrine levels in nine patients anesthetized with 1.1% isoflurane in 50% nitrous oxide and oxygen were studied. End-tidal carbon dioxide (CO2) was maintained at 34 +/- 2 mmHg. Cardiac output (CO) and mean arterial pressure (MAP) were measured, and total peripheral resistance (TPR) was calculated at the following time points: (1) after immersion prior to shock wave therapy (control); (2) after 300 shocks; (3) after 800 shocks; and (4) 5 minutes after the completion of ESWL with the patient still immersed. Circulating epinephrine and norepinephrine concentrations were determined at the above times as well as before and after induction of anesthesia but prior to immersion. There was a statistically significant (p less than 0.05) decrease in CO and an increase (p less than 0.05) in MAP and TPR with ESWL treatment. These values returned to baseline levels when treatment was stopped. Plasma epinephrine and norepinephrine values did not change significantly throughout the study period. It was concluded that these ESWL-associated hemodynamic changes were probably not mediated via epinephrine or norepinephrine. |
| Databáze: | OpenAIRE |
| Externí odkaz: |
|