MOESM2 of Enzymatic and molecular characterization of insecticide resistance mechanisms in field populations of Aedes aegypti from Selangor, Malaysia

Autor: Cherng-Shii Leong, Vythilingam, Indra, Liew, Jonathan, Meng-Li Wong, Wan-Yusoff, Wan, Yee-Ling Lau
Rok vydání: 2019
DOI: 10.6084/m9.figshare.8142686
Popis: Additional file 2: Figure S1. Gel electrophoresis of AS-PCR products corresponding to the Ae. aegypti sodium channel gene mutation. a F1534C mutation: each of the three genotypes is shown. Lane 1: ultra-low range DNA ladder; Lane 2: wild-type homozygous (FF); Lane 3: heterozygous (FC); Lane 4: mutant homozygous (CC); Lane 5: negative control. b V1016G mutation: Lane 1: ultra-low range DNA ladder marker; Lane 2: mutant homozygous (GG); Lane 3: heterozygous (VG); Lane 4: wild-type homozygous (VV); Lane 5: negative control. c S989P mutation: Lane 1: 100 bp DNA ladder marker; Lanes 2, 3: wild-type homozygous (SS); Lanes 4, 5: heterozygous (SP); Lanes 6, 7: mutant homozygous (PP); Lanes 8, 9: negative control. Figure S2. Genotype sequence of V1016G mutation. Figure S3. Genotype sequence of S989P mutation. Figure S4. Genotype sequence of F1534C mutation.
Databáze: OpenAIRE