Sensorineural hearing loss in patients with cerebral palsy after asphyxia and hyperbilirubinemia
| Autor: | Eiji Kitazumi, Masaki Sano, Kazuo Kodama, Kimitaka Kaga |
|---|---|
| Rok vydání: | 2005 |
| Předmět: |
Adult
Male medicine.medical_specialty Adolescent Hearing loss Hearing Loss Sensorineural Otoacoustic Emissions Spontaneous Otoacoustic emission Audiology Cerebral palsy otorhinolaryngologic diseases medicine Humans Child Cochlear Nerve Organ of Corti Hyperbilirubinemia Asphyxia Asphyxia Neonatorum medicine.diagnostic_test business.industry Cerebral Palsy Infant Newborn Cochlear nerve Auditory Threshold General Medicine medicine.disease Magnetic Resonance Imaging Audiometry Evoked Response Hair Cells Auditory Outer medicine.anatomical_structure Otorhinolaryngology Child Preschool Anesthesia Pediatrics Perinatology and Child Health Audiometry Pure-Tone Sensorineural hearing loss sense organs medicine.symptom Audiometry business |
| Zdroj: | International Journal of Pediatric Otorhinolaryngology. 69:1211-1217 |
| ISSN: | 0165-5876 |
| DOI: | 10.1016/j.ijporl.2005.03.014 |
| Popis: | The location of lesions causing hearing loss in patients with cerebral palsy due to asphyxia or neonatal hyperbilirubinemia has remained unclear. We performed behavioral audiometry, distortion product otoacoustic emission (DPOAE) and auditory brainstem evoked response (ABR) in six patients with cerebral palsy due to asphyxia or neonatal hyperbilirubinemia in order to determine the lesion location causing their hearing impairment. In all cases, behavioral audiometry revealed a threshold elevation of 50-75 dB and ABR were no response. DPOAE were totally absent in five patients and normal in one patient. Our study suggests that lesions causing hearing loss potentially include the organ of Corti especially at the outer hair cells and the cochlear nerve. |
| Databáze: | OpenAIRE |
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