Supraventricular tachycardia in a patient with Lown-Ganong-Levine syndrome associated with apical hypertrophic cardiomyopathy
| Autor: | Yoichi Imamura, Junichi Inoue, Hiroshi Nakashima, Kuniaki Fukuyama, Motonobu Hayano, Mitsuhiro Tsuruta, Katsusuke Yano, Shinsuke Tsuji, Yoshiki Eguchi, Shuzo Matsuo |
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| Rok vydání: | 1988 |
| Předmět: |
Tachycardia
Cardiac Catheterization medicine.medical_specialty Pre-Excitation Syndromes Cardiomyopathy Electrocardiography T wave Internal medicine Tachycardia Supraventricular medicine Humans cardiovascular diseases PR interval Lown–Ganong–Levine syndrome medicine.diagnostic_test business.industry Hypertrophic cardiomyopathy Lown-Ganong-Levine Syndrome Cardiomyopathy Hypertrophic Middle Aged medicine.disease Echocardiography cardiovascular system Cardiology Female Supraventricular tachycardia medicine.symptom Cardiology and Cardiovascular Medicine business |
| Zdroj: | Japanese Heart Journal. 29:249-256 |
| ISSN: | 1348-673X 0021-4868 |
| Popis: | Electrophysiologic study of a 55-year-old patient with Lown-Ganong-Levine syndrome associated with apical hypertrophic cardiomyopathy is reported. The patient had a history of recurrent attacks of tachyarrhythmia and his electrocardiogram showed a short P-R interval (0.10 sec) with narrow QRS complex and left ventricular hypertrophy with giant negative T waves. His cineangiogram showed severe apical hypertrophy. An electrophysiologic study was performed. The results of programmed atrial pacing show the existence of the dual A-V nodal pathways. The A-H interval at rapid atrial pacing increased maximally by 103 msec. Atrial stimulation could depolarize parts of the atrium without altering the supraventricular tachycardia. These findings suggested that preferential rapidly conducting A-V nodal and intranodal reentry are the responsible mechanisms in this reciprocating tachycardia. We conclude that the short P-R interval was due to intranodal reentry through the dual A-V nodal pathways. To our knowledge, a case of Lown-Ganong-Levine syndrome with apical hypertrophic cardiomyopathy has not been previously described in the literature. |
| Databáze: | OpenAIRE |
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