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Landry in 1859 described an acutely progressive paralysis with slight or no sensory involvements and negative microscopic observations, noting recovery in some mild cases. The Oppenheim textbooks of 1911 and 1923 accept the syndrome as such, emphasizing the rapid onset of a flaccid paralysis without pain or especial tenderness and usually without marked disturbances of sensibility, death usually resulting from bulbar paralysis but recovery occurring not infrequently. A close genetic relationship to acute anterior poliomyelitis and polyneuritis is recognized, as well as the fact that inflammatory and vascular changes occur almost always in the cord.Purves-Stewart1 treats the syndrome throughout as Landry's paralysis, a condition of the lower motor neuron attributable to various infective agents. Wechsler2 mentions a Landry type of poliomyelitis, and again a syndrome, usually without fever and probably toxico-infectious in character, in which recovery may occur. Jelliffe and White3 view it as belonging primarily |
