Surgical Management of CME Associated with Vitreoretinal Interface

Autor: Javier Elizalde, J. Fernando Arevalo, Juliana Bottós, Emerson Badaro, Mauricio Maia
Rok vydání: 2016
Předmět:
Zdroj: Cystoid Macular Edema ISBN: 9783319397641
DOI: 10.1007/978-3-319-39766-5_11
Popis: Cystoid macular edema (CME) after ocular surgery was first described in 1953 by Irvine and is related to the release of inflammatory mediators such as prostaglandins, leukotrienes, histamine, bradykinins, platelet-activating factor (PAF), and interleukin (IL)-1 [1–4]. The original report described cystoid changes after vitreous incarceration at the corneal wound after intracapsular cataract surgery. However, nowadays, this terminology is used for any macular edema after surgical procedures. A classic feature of this disease is the cystoid macular abnormalities related to swelling of the outer plexiform layer following release of cytokines at the vitreous cavity, which results in the classic hallmark petaloid pattern seen by fluorescein angiogram (FA) (Fig. 1); additionally, new insights have been added by spectral-domain optical coherence tomography (OCT) technology (Fig. 1b). The management of this entity is described in another chapter. The differential diagnosis should include epiretinal membrane, macular hole, age-related macular degeneration, central serous chorioretinopathy, and, most importantly, vitreomacular traction (VMT) syndrome [1–4].
Databáze: OpenAIRE