Surgical Management of CME Associated with Vitreoretinal Interface
Autor: | Javier Elizalde, J. Fernando Arevalo, Juliana Bottós, Emerson Badaro, Mauricio Maia |
---|---|
Rok vydání: | 2016 |
Předmět: |
medicine.medical_specialty
genetic structures business.industry medicine.medical_treatment Vitreomacular traction Macular degeneration Cataract surgery medicine.disease eye diseases Serous fluid Ophthalmology medicine sense organs Differential diagnosis Epiretinal membrane business Macular edema Macular hole |
Zdroj: | Cystoid Macular Edema ISBN: 9783319397641 |
DOI: | 10.1007/978-3-319-39766-5_11 |
Popis: | Cystoid macular edema (CME) after ocular surgery was first described in 1953 by Irvine and is related to the release of inflammatory mediators such as prostaglandins, leukotrienes, histamine, bradykinins, platelet-activating factor (PAF), and interleukin (IL)-1 [1–4]. The original report described cystoid changes after vitreous incarceration at the corneal wound after intracapsular cataract surgery. However, nowadays, this terminology is used for any macular edema after surgical procedures. A classic feature of this disease is the cystoid macular abnormalities related to swelling of the outer plexiform layer following release of cytokines at the vitreous cavity, which results in the classic hallmark petaloid pattern seen by fluorescein angiogram (FA) (Fig. 1); additionally, new insights have been added by spectral-domain optical coherence tomography (OCT) technology (Fig. 1b). The management of this entity is described in another chapter. The differential diagnosis should include epiretinal membrane, macular hole, age-related macular degeneration, central serous chorioretinopathy, and, most importantly, vitreomacular traction (VMT) syndrome [1–4]. |
Databáze: | OpenAIRE |
Externí odkaz: |