Popis: |
The ability of bacterial endotoxins (ET) to alter the course of experimental infections is well-known. The transient increase in homologous and heterologous resistance that develops within hours after ET administration may be related to enhanced macrophage and reticuloendothelial activity (1, 2) and/or to enhancement of preexisting specific antibody titres according to published observations (3, 4). Furthermore, it has been suggested that at least a part of the biological activities provoked by ET are based on a hypersensitivity state of the host (5, 6). |