HTAP2 multi-model estimates of premature human mortality due to intercontinental transport of air pollution
Autor: | R. Bradley Pierce, Ulas Im, Johannes Flemming, Allen J. Lenzen, Takashi Sekiya, Terry Keating, J. Jason West, Huisheng Bian, Louisa K. Emmons, Xiaohua Pan, Raquel A. Silva, Jan Eiof Jonson, Mian Chin, Daven K. Henze, Kengo Sudo, Rokjin J. Park, Frank Dentener, Ciao-Kai Liang, Tom Kucsera, Toshihiko Takemura, Marianne Tronstad Lund, Gerd A. Folberth, Meiyun Lin, Yanko Davila |
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Rok vydání: | 2018 |
Předmět: |
Pollution
Pollutant Ozone 010504 meteorology & atmospheric sciences Ambient air pollution Chemical transport model media_common.quotation_subject Air pollution 010501 environmental sciences Atmospheric sciences medicine.disease_cause 01 natural sciences chemistry.chemical_compound chemistry medicine Environmental science East Asia Air quality index 0105 earth and related environmental sciences media_common |
DOI: | 10.5194/acp-2017-1221 |
Popis: | Ambient air pollution from ozone and fine particulate matter is associated with premature mortality. As emissions from one continent influence air quality over others, changes in emissions can also influence human health on other continents. We estimate global air pollution-related premature mortality from exposure to PM2.5 and ozone, and the avoided deaths from 20 % anthropogenic emission reductions from six source regions, North America (NAM), Europe (EUR), South Asia (SAS), East Asia (EAS), Russia/Belarus/Ukraine (RBU) and the Middle East (MDE), three emission sectors, Power and Industry (PIN), Ground Transportation (TRN) and Residential (RES) and one global domain (GLO), using an ensemble of global chemical transport model simulations coordinated by the second phase of the Task Force on Hemispheric Transport of Air Pollution (TF-HTAP2), and epidemiologically-derived concentration-response functions. We build on results from previous studies of the TF-HTAP by using improved atmospheric models driven by new estimates of 2010 emissions, with more source and receptor regions, new consideration of source sector impacts, and new epidemiological mortality functions. We estimate 290,000 (95 % CI: 30,000, 600,000) premature O3-related deaths and 2.8 million (0.5 million, 4.6 million) PM2.5-related premature deaths globally for the baseline year 2010. While 20 % emission reductions from one region generally lead to more avoided deaths within the source region than outside, reducing emissions from MDE and RBU can avoid more O3-related deaths outside of these regions than within, and reducing MDE emissions also avoids more PM2.5-related deaths outside of MDE than within. In addition, EUR, MDE and RBU have more avoided O3-related deaths from reducing foreign emissions than from domestic reductions. For six regional emission reductions, the total avoided extraregional mortality is estimated as 10,300 (6,700, 13,400) deaths/year and 42,000 (12,400, 60,100) deaths/year through changes in O3 and PM2.5, respectively. Interregional transport of air pollutants leads to more deaths through changes in PM2.5 than in O3, even though O3 is transported more on interregional scales, since PM2.5 has a stronger influence on mortality. In sectoral emission reductions, TRN emissions account for the greatest fraction (26–53 % of global emission reduction) of O3-related premature deaths in most regions, except for EAS (58 %) and RBU (38 %) where PIN emissions dominate. In contrast, PIN emission reductions have the greatest fraction (38–78 % of global emission reduction) of PM2.5-related deaths in most regions, except for SAS (45 %) where RES emission dominates. The spread of air pollutant concentration changes across models contributes most to the overall uncertainty in estimated avoided deaths, highlighting the uncertainty in results based on a single model. Despite uncertainties, the health benefits of reduced intercontinental air pollution transport suggest that international cooperation may be desirable to mitigate pollution transported over long distances. |
Databáze: | OpenAIRE |
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