The Effect of Captopril on Urinary Kinins and Urinary Kallikrein Activity in Essential Hypertension

Autor:	Margareta Bramnert, J. F. Dymling, U. L. Hulthén, B. Hökfelt
Rok vydání:	1981
Předmět:	chemistry.chemical_classification medicine.medical_specialty business.industry Urinary system Captopril Vasodilation Essential hypertension medicine.disease Angiotensin II Natriuresis Enzyme Blood pressure Endocrinology chemistry Internal medicine medicine business medicine.drug
Zdroj:	Frontiers in Hypertension Research ISBN: 9781461259015
DOI:	10.1007/978-1-4612-5899-5_88
Popis:	Angiotensin-Converting enzyme is most likely identical to kininase II (1). This enzyme generates angiotensin II, the most potent vasoconstrictor known (2) By degrading kinins as well, which are potent vasodilators (3), and increasing natriuresis (4), it may be that it increases blood pressure by two separate mechanisms (Fig. 1).
Databáze:	OpenAIRE
Externí odkaz:	https://explore.openaire.eu/search/publication?articleId=doi_________::99aa7b5e6c63640b59a3c9c7de979a61 https://doi.org/10.1007/978-1-4612-5899-5_88 Zobrazit plný text záznamu