| Popis: |
With the advent of thrombolytic therapy for acute ischemic stroke it may soon be possible to rapidly restore blood flow to an ischemic area of the brain. However, reperfusion may carry its own attendant risk of further tissue injury. There is an increasing body of evidence which suggests that neutrophils play an important role in mediating tissue damage during acute ischemic stroke [1, 2, 4, 5]. Moreover, there are suggestions that neutrophils may be at least partially responsible for the “no reflow” phenomenon [3]. |
