Increased Hindlimb Leukocyte Concentration in a Chronic Rodent Model of Venous Hypertension

Autor:	Hahn, Tara L., Unthank, Joseph L., Lalka, Stephen G.
Zdroj:	Journal of Surgical Research; January 1999, Vol. 81 Issue: 1 p38-41, 4p
Abstrakt:	Background.The pathophysiologic mechanism for tissue damage in chronic venous insufficiency (CVI) is venous hypertension (VH), the primary mediator behind leukocyte trapping in tissues. We developed a new rodent model of chronic hindlimb VH to allow testing of the microvascular dysfunction that occurs in clinical CVI.
Databáze:	Supplemental Index
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