| Autor: |
Day, Rufus S., Honore, Louis H., Dobler, Kelly D. |
| Zdroj: |
Mutation Research/DNA Repair; March 1991, Vol. 254 Issue: 2 p153-160, 8p |
| Abstrakt: |
With increasing passage in culture, the human fibroblast cell strain GM11 lost the Mer+phenotype (the ability to support the growth of adenovirus 5 damaged prior to infection by MNNG). All of 46 embryonic strains prepared either from various organs of 20 fetuses from 6–7 to 13–14 weeks of gestational age or from two hydatidiform moles showed normal repair of MNNG-treated virus. We conclude that human fetal strains are not usually deficient in such repair, and that the behavior of GM11 is atypical. |
| Databáze: |
Supplemental Index |
| Externí odkaz: |
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