| Autor: |
Scanlon, Michael J., Myers, Alan M., Schneeberger, Richard G., Freeling, Michael |
| Zdroj: |
The Plant Journal; October 1997, Vol. 12 Issue: 4 p901-909, 9p |
| Abstrakt: |
The defective kernel mutation empty pericarp2‐R(emp2‐R) causes retardation and subsequent abortion of maize kernel development. Analyses of genetic aneuploid kernels indicate that the embryo phenotype is not dependent on the endosperm genotype; the mutation conditions embryo defects even in the presence of a normal endosperm. Embryos reach an abnormal coleoptilar stage before aborting and disintegrating. The mutants form primary embryonic organs only; the scutellum and coleoptile develop, but no leaves are formed. Immunohisto‐localization studies utilized KNOX homeodomain proteins as markers of meristem formation and identity. These analyses indicate that the shoot meristem forms in emp2‐Rmutant embryos, but does not mature to a tunica‐corpus shape. No evidence of leaf founder cell initialization was revealed in the mutant meristems. These data indicate that the emp2gene may be required for embryogenic patterning beyond the coleoptilar stage of development. |
| Databáze: |
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