Autor:	Khomullo, G. V.
Zdroj:	Bulletin of Experimental Biology and Medicine; March 1962, Vol. 53 Issue: 2 p211-214, 4p
Abstrakt:	Bone tissue regeneration after fracture of the tibia was studied in albino rats under conditions of different BMR levels. Thyroidin caused a marked rise of the BMR and accelerated the regeneration of bone: mesenchymal cells exhibited increased activity, large numbers of ostcoblasts in the regenerating area accumulated more rapidly, and the replacement of cartilagenous by osteoid tissue was accelerated. A low BMR caused by 6-methylthiouracil retarded bone regeneration.
Databáze:	Supplemental Index
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