Autor: |
Shiono, Noritsugu, Rao, Vivek, Weisel, Richard D., Kawasaki, Muneyasu, Ren-Ke Li, Mickle, Donald A.G., Fedak, Paul W.M., Tumiati, Laura C., Ko, Lawrence, Verma, Subodh |
Předmět: |
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Zdroj: |
American Journal of Physiology: Heart & Circulatory Physiology; Mar2002, Vol. 282 Issue 3, pH805, 11p, 6 Black and White Photographs, 4 Diagrams, 1 Chart, 9 Graphs |
Abstrakt: |
Presents a study that found that low-volume anoxia and reoxygenation increased nitric oxide synthase (NOS) activity and facilitated the conversion of L-arginine to nitric oxide (NO) to protect against heart cell injury. Measurement of NO production; Characterization of NOS isoforms in human heart cells after low-volume anoxia and reoxygenation; Effects of L-arginine on cellular injury and NO production after low-volume anoxia and reoxygenation. |
Databáze: |
Complementary Index |
Externí odkaz: |
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