| Abstrakt: |
A recent study conducted by the Dermatology Service in Brooklyn, New York, explores the role of Th17 cells and interleukin (IL)-17 in the pathogenesis of keloids. Keloids are characterized by excessive fibroblast proliferation and connective tissue deposition, causing physical disfigurement, pain, anxiety, and depression. The study found that Th17 cells and IL-17 play a significant role in the inflammatory and fibrotic processes of keloid formation. Targeting the IL-17 pathway could potentially offer a new therapeutic approach for improving outcomes in keloid patients. Further research is needed to fully understand the role of Th17 cells and IL-17 in keloid pathogenesis and to assess the safety and efficacy of targeting this pathway in human studies. [Extracted from the article] |
