Abstrakt: |
A study conducted by Fudan University in Shanghai, China, explored the role of the RNA-binding protein RBM15 in myocardial infarction (MI), or heart attack. The study found that RBM15 plays a significant role in stabilizing a target related to cell apoptosis, which may provide new insights into MI therapy. The research used methylated RNA immunoprecipitation sequencing to examine the differences in N6-methyladenosine (m6A) levels between MI and normal tissues. The findings suggest that elevated m6A levels act as a protective mechanism in MI, and RBM15 is significantly upregulated in MI and promotes cardiac function. [Extracted from the article] |