New Data from Ningxia Medical University Illuminate Research in Erectile Dysfunction (Nesfatin-1 regulates the phenotype transition of cavernous smooth muscle cells by activating PI3K/AKT/mTOR signaling pathway to improve diabetic erectile...).

Předmět:	PHENOTYPIC plasticity IMPOTENCE MUSCLE cells SMOOTH muscle CELLULAR signal transduction
Zdroj:	Diabetes Week; 7/8/2024, p189-189, 1p
Abstrakt:	New research from Ningxia Medical University in China explores the impact of Nesfatin-1 on type 2 diabetic erectile dysfunction (T2DMED) and its underlying mechanism in regulating the phenotypic switching of corpus cavernosum smooth muscle cells (CCSMCs). The study found that Nesfatin-1 effectively improved body weight, blood glucose levels, and erectile function in diabetic mice. It also regulated the phenotypic switching of CCSMCs by activating the PI3K/AKT/mTOR signaling pathway. These findings suggest that Nesfatin-1 may be a potential treatment for diabetic erectile dysfunction. [Extracted from the article]
Databáze:	Complementary Index
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