| Abstrakt: |
Background: Cardiovascular illnesses, such as acute myocardial infarction and angina pectoris, are among the most common health problems in the general population and important public health concerns in developing countries. The pathophysiology of periodontal problems, which are caused by bacteria that interact with the human immune system, may explain the link between CVD and chronic periodontitis (CP). Methods: A total of 100 subjects diagnosed with coronary artery disease were placed into four different groups with 25 each. Inflammatory biomarkers such as CRP and WBC count were measured, as well as cardiac biomarkers such as Troponin I and cardiovascular risk factors such as lipid profile (VLDL, LDL, HDL, TC) and periodontal parameters such as PI, GI, PPD, CAL, PESA, and PISA were recorded. Results: Statistically significant relationships between PI, GI, mean PPD, mean CAL and cTnI, LDL, and TC were observed. PESA and PISA levels were statistically significant with LDL and TC. Positive relations were seen among various groups with periodontal parameters, cardiac biomarkers, and risk factors such as WBC COUNT, CRP, VLDL, and HDL. Conclusion: Periodontal disease has significant physiological effects similar to the pathogenesis of coronary heart disease. Biomarker changes have been measured to support these pathological actions. Patients with periodontal disease are at higher risk of developing coronary heart disease. [ABSTRACT FROM AUTHOR] |
