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Zdroj: |
Genomics & Genetics Weekly; 1/12/2024, p1590-1590, 1p |
Abstrakt: |
A recent study conducted by researchers at the Medical Faculty and University Hospital Duesseldorf in Germany has found that nicotine exposure can potentially alter endothelial inflammation and cell adhesion through the regulation of LGALS9 (Galectin-9). The study focused on human aortic endothelial cells (HAoECs) and used RNA sequencing (RNA-Seq) to identify genes that were regulated following nicotine exposure. The researchers concluded that LGALS9 may serve as a key regulator for essential endothelial cell processes and could be a potential target in the development of aortic pathologies. This research provides valuable insights into the effects of nicotine on cardiovascular health. [Extracted from the article] |
Databáze: |
Complementary Index |
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