| Abstrakt: |
A research study conducted at the Affiliated Hospital of Jiujiang University in China explores the mechanisms and therapeutic targets of mitophagy after intracerebral hemorrhage. Mitochondria, responsible for cellular energy production, also play a role in calcium homeostasis, damaged organelle clearance, signaling, and cell survival. The study suggests that mitochondrial damage and autophagy are crucial for neuronal metabolism and functional recovery after intracerebral hemorrhage, and are closely linked to inflammatory responses, oxidative stress, apoptosis, and other pathological processes. The research investigates the neuroprotective role of mitophagy through various approaches, including normobaric oxygen therapy. [Extracted from the article] |
