Oxidative stress in atherosclerosis-prone mouse is due to low antioxidant capacity of mitochondria.

Autor:	Oliveira, Helena C. F., Cosso, Ricardo G., Alberici, Luciane C., Maciel, Evelise N., Salerno, Alessandro G., Dorighello, Gabriel G., Velho, Jesus A., De Faria, Eliana C., Vercesi, Aníbal E.
Předmět:	MITOCHONDRIA REACTIVE oxygen species ATHEROSCLEROSIS LOW density lipoproteins LABORATORY mice PYRIDINE nucleotides TRIGLYCERIDES CHOLESTEROL
Zdroj:	FASEB Journal; Feb2005, Vol. 19 Issue 2, p278-280, 3p, 1 Diagram, 2 Graphs
Abstrakt:	Presents a study which examined the mitochondrial function and production of reactive oxygen species in the atherosclerosis-susceptible, hypercholesterolemic low density lipoprotein (LDL) receptor knockout mice. Results of the isolation of mitochondria from the LDL mice; Oxidizing of pyridine nucleotides in LDL receptor knockout mouse; Secretion and synthesis of triglycerides and cholesterol in the livers of hypercholesterolemic low density lipoprotein (LDL) receptor knockout mice.
Databáze:	Complementary Index
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