Exploring Target Site Mutation in a Glufosinate-Resistant Palmer Amaranth Accession from Arkansas.

Autor: González-Torralva, F., Norsworthy, J. K., Carvalho-Moore, P., Priess, G. L., Piveta, L. B., Barber, L. T., Butts, T. R.
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Zdroj: B.R. Wells Rice Research Studies - Arkansas Agricultural Experiment Station, University of Arkansas System; Dec2021, Issue 680, p137-139, 3p
Abstrakt: Palmer amaranth (Amaranthus palmeri S. Watson) is a very difficult to control weed in different cropping systems including cotton (Gossypium hirsutum L.) and soybean [Glycine max (L.) Merr.]. Different chemistries have been used to control it, gradually leading to a loss of susceptibility. A Palmer amaranth accession was collected in eastern Arkansas after the failure of glufosinate to effectively control this weed in cotton. Under greenhouse conditions, the application of glufosinate at the recommended field rate (1×, 32 fl oz/ac) has not been effective. The objective of this research was to elucidate if single nucleotide polymorphisms are involved in the recently documented resistance within this accession. For that purpose, the glutamine synthetase (GS) gene was sequenced following standard procedures. Comparison of the nucleotides and their deduced protein between resistant and susceptible accessions have revealed no amino acid substitution at position 171, the site where resistance has occurred in other weeds. The outcome of this research suggests that the resistant accession most likely has a resistance mechanism other than a target-site mutation that leads to the ineffectiveness of glufosinate on this population. [ABSTRACT FROM AUTHOR]
Databáze: Complementary Index