Alexia without agraphia: from infarctions to malignancies.

Autor:	Jauregui R; Department of Neurology, NYU Grossman School of Medicine, New York, New York, USA rjaureg91@gmail.com., Greenberg J; Department of Neurology, NYU Grossman School of Medicine, New York, New York, USA., Kuball P; Department of Neurology, NYU Grossman School of Medicine, New York, New York, USA., Newbold DJ; Department of Neurology, NYU Grossman School of Medicine, New York, New York, USA., Patel R; Department of Neurology, NYU Grossman School of Medicine, New York, New York, USA., Staudinger R; Department of Neurology, NYU Grossman School of Medicine, New York, New York, USA.; Department of Neurology, Veterans Affairs New York Harbor Healthcare System, New York, New York, USA.
Jazyk:	angličtina
Zdroj:	Practical neurology [Pract Neurol] 2024 Sep 23. Date of Electronic Publication: 2024 Sep 23.
DOI:	10.1136/pn-2024-004235
Abstrakt:	Alexia without agraphia is a neurological syndrome characterised by an acquired inability to read with a preserved ability to write. It is caused by the combined effect of two lesions: in the splenium of the corpus callosum and in the occipital lobe of the dominant hemisphere. Splenial lesions disconnect the language areas in the temporal and parietal lobes of the dominant hemisphere from the visual areas in the occipital cortex of the contralateral side, while lesions in the dominant occipital lobe cause homonymous hemianopia. We describe two patients with lesions affecting the splenium and dominant occipital lobe, with different causes. Together, these cases highlight the importance of performing a thorough language evaluation in patients presenting with homonymous visual field deficits, as otherwise, clinicians may overlook impairments in writing (agraphia) or reading (alexia). Competing Interests: Competing interests: None declared. (© Author(s) (or their employer(s)) 2024. No commercial re-use. See rights and permissions. Published by BMJ.)
Databáze:	MEDLINE
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