β-Adrenergic Agonist-Induced Lactic Acidosis: A Case Report.
| Autor: | Blush RR 3rd; Raymond R. Blush III is Clinical Assistant Professor, University of Michigan School of Nursing, Room 2304, 400 SNB N Ingalls, Ann Arbor, MI 48109 (rayblush@umich.edu). |
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| Jazyk: | angličtina |
| Zdroj: | AACN advanced critical care [AACN Adv Crit Care] 2024 Sep 15; Vol. 35 (3), pp. 222-227. |
| DOI: | 10.4037/aacnacc2024787 |
| Abstrakt: | β-Adrenergic agonist medications such as albuterol are the mainstay for treatment of patients with acute asthma exacerbations. Patients who present to the emergency department with severe symptoms are often treated with multiple albuterol doses in sequence to maximize the impact of the medications, relax bronchoconstriction, and relieve their breathlessness. Patients who present with acute dyspnea have numerous potential causes of hyperlactatemia and acidosis including an uncommonly recognized outcome of albuterol administration. This clinical case report outlines a scenario where a patient who was treated for an acute asthma exacerbation had rising lactate levels despite improving clinically. Causes of elevated lactate levels are discussed, particularly related to β-adrenergic agonist use, and considerations for monitoring and withdrawal of albuterol administration are outlined. (©2024 American Association of Critical-Care Nurses.) |
| Databáze: | MEDLINE |
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