Author Correction: Myt1l safeguards neuronal identity by actively repressing many non-neuronal fates.

Autor: Mall M; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA., Kareta MS; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA.; Genetics and Genomics Group, Sanford Research, Sioux Falls, SD, USA., Chanda S; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA.; Department of Molecular and Cellular Physiology and Howard Hughes Medical Institute, Stanford University, Stanford, California, USA., Ahlenius H; Department of Clinical Sciences, Division of Neurology and Lund Stem Cell Center, Lund University, Lund, Sweden., Perotti N; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA., Zhou B; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA.; Department of Molecular and Cellular Physiology and Howard Hughes Medical Institute, Stanford University, Stanford, California, USA., Grieder SD; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA., Ge X; Department of Developmental Biology, Stanford University, Stanford, California, USA.; Molecular and Cellular Biology, University of California Merced, Merced, CA, USA., Drake S; Department of Clinical Sciences, Division of Neurology and Lund Stem Cell Center, Lund University, Lund, Sweden., Ang CE; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA., Walker BM; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA., Vierbuchen T; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA.; Department of Neurobiology, Harvard Medical School, Boston, Massachusetts, USA., Fuentes DR; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA., Brennecke P; Department of Genetics, Stanford University, Stanford, California, USA.; Leibniz-Institute for Molecular Pharmacology, Berlin, Germany., Nitta KR; Division of Functional Genomics and Systems Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institutet, Stockholm, Sweden.; Division of Genomic Technologies, RIKEN Center for Life Science Technologies, Yokohama, Japan., Jolma A; Division of Functional Genomics and Systems Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institutet, Stockholm, Sweden., Steinmetz LM; Department of Genetics, Stanford University, Stanford, California, USA.; Genome Biology Unit, European Molecular Biology Laboratory (EMBL), Heidelberg, Germany., Taipale J; Division of Functional Genomics and Systems Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institutet, Stockholm, Sweden.; Genome Scale Biology Program, University of Helsinki, Helsinki, Finland., Südhof TC; Department of Molecular and Cellular Physiology and Howard Hughes Medical Institute, Stanford University, Stanford, California, USA., Wernig M; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California, USA. wernig@stanford.edu.
Jazyk: angličtina
Zdroj: Nature [Nature] 2024 Jun; Vol. 630 (8017), pp. E11.
DOI: 10.1038/s41586-024-07594-8
Databáze: MEDLINE