| Autor: |
Popa GL; Department of Microbiology, 'Carol Davila' University of Medicine and Pharmacy, 020021 Bucharest, Romania., Popa MI; Department of Microbiology, 'Carol Davila' University of Medicine and Pharmacy, 020021 Bucharest, Romania.; 'Cantacuzino' National Military Medical Institute for Research and Development, 011233 Bucharest, Romania. |
| Jazyk: |
angličtina |
| Zdroj: |
Microorganisms [Microorganisms] 2022 Jun 21; Vol. 10 (7). Date of Electronic Publication: 2022 Jun 21. |
| DOI: |
10.3390/microorganisms10071265 |
| Abstrakt: |
In recent years, the role of oxidative stress has been investigated in an increasing number of infections. There is a close link between the inflammation that accompanies infections and oxidative stress. Excessive reactive oxygen species induce harmful effects on cell components, including lipids, proteins, and nucleic acids. A growing body of evidence attests to the role of oxidative stress in the pathogenesis of viral liver infections, especially in hepatitis C virus (HCV) infection. Regarding hepatitis B virus (HBV) infection, the data are limited, but important progress has been achieved in recent years. This review presents the latest advances pertaining to the role of the oxidative stress byproducts in the pathogenesis of chronic hepatitis B, constituting a source of potential new markers for the evaluation and monitoring of patients with chronic hepatitis B. |
| Databáze: |
MEDLINE |
| Externí odkaz: |
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